AAT Phase 1 is focused primarily upon the unconscious survival reflexes that are located in the Brain Stem (first brain) that are triggered by our instinctive reactions to sensory stimuli that the brain perceives as a threat. It does not matter if the threat is real or perceived. The first brain will ALWAYS react in a way that attempts to ensure the survival of the human organism. When these reactions are coupled with a potential life threat, the physical implications are profound and often become chronic, leaving many patients with nothing but a sense of hopelessness. AAT Phase 1 can break these conditioned patterns of chronicity by communicating with the Amygdala and subsequently the brain stem, restoring homeostasis to all systems of the body.

1. Understand the Clinical implications of the ANS in Chronic Conditions
2. Understand the Complex Relationships between the Brainstem, Limbic System and Neocortex
3. Understand the Roles of Extrinsic and Intrinsic Memory Systems within Chronic Pain Patterns
4. Understand How Mechanoreceptors Communicate with the Somatosensory Cortex to Eliminate Chronic Tension and Pain Patterns
5. Learn the Disorders of the Sympathetic and Parasympathetic Nervous Systems
6. Learn How Stress Creates Physiological Changes Within Multiple Body Systems
7. Learn to Implement Treatment Interventions Specifically Targeting the ANS

AAT™ Phase 2 will build on the success you have had with your patients during AAT™ Phase 1, or it may help those patients that had partial relief during Phase 1. AAT™ Phase 2 targets the Limbic System (Emotional Brain or Second Brain) and will help to release negative emotional associations that activate habitual fight or flight patterns from the chronic cycles that our patients/clients experience. We will discuss the science that supports our current understanding of the Limbic System and how it interfaces with the Brain Stem (first brain) to create chronic patterns. We are fortunate to be able to utilize members of our scientific and medical advisory board to assist in our understanding of the second brain level from a theoretical, practical and clinical perspective.

AAT™ Phase 3 is designed to disconnect our experiential associations and anticipatory expectations that facilitate rapid cycling and ultimately fuels our chronic patterns, making homeostasis very difficult (if not impossible) to achieve. This occurs in the Neo-cortex (Third Brain) where our conscious associations, memories and thoughts have become patterned to trigger the fight or flight response and perpetuate your patient's patterns of chronicity. We will discuss the relationship between the first, second and third brains and teach methods to restore homeostasis to the brain and body. Once again, we will utilize members of our scientific and medical advisory board to assist in understanding this brain level and how all 3 levels of the brain interact in an unbalanced fashion to perpetuate these painful and debilitating cycles of dysfunction.

AAT™ Phase 2 and 3 Course Objectives

1. Review the brainstem (first brain) impact on chronic pain cycles
2. Understand the meaning of the terms Association and Awareness in the context of AAT™
3. Understand how the limbic system (second brain) functions and interacts with the first and third brain in situations of chronicity
4. Understand how the neocortex (third brain) functions and interacts with the first and second brain in situations of chronicity
5. Understand the concept of rapid cycling and the importance of creating homeostasis in order to create lasting changes
6. Learn how to access the limbic system to disconnect the physical manifestations of our emotional experiences and how to teach our patients how to relegate these emotions to memories only.
7. Learn how to access the neocortex and how to teach our patients about associative awareness in the context of their own situations of chronicity
8. Reiterate the importance of self regulation in respect to all three levels of the brain in order to disrupt the vicious chronic cycles and to ensure proper implementation of neuroplastic changes resulting in classically re-conditioning of the Brain to maintain neurochemical homeostasis.